Abstract
Introduction
The aim of this retrospective study was to examine the effect of orthodontic extrusion
on the pulpal vitality of maxillary incisors with a history of trauma.
Methods
Pulpal condition was examined clinically (rating of crown color and sensitivity testing
with a cryogenic spray) and radiologically (periapical and panoramic radiographs)
after orthodontic extrusion of previously traumatized (Orthodontics/Trauma group,
n = 77) and nontraumatized teeth (Orthodontics group, n = 400) and after previous
dental trauma without subsequent orthodontic treatment (Trauma group, n = 193). Dental
traumata were divided into hard tissue injuries (fracture of enamel and enamel chipping,
fracture of enamel-dentin without pulpal involvement, fracture of enamel-dentin with
pulpal involvement, root fracture, crown-root fracture) and periodontal injuries (concussion,
subluxation, intrusion, extrusion, lateral luxation, and avulsion).
Results
Teeth in the Orthodontics/Trauma group showed a significantly higher frequency of
pulp necrosis than teeth in the Orthodontics group (P < .001) or teeth in the Trauma group (P < .009). In addition, teeth in the Orthodontics/Trauma group with periodontal injuries
showed a significantly higher rate of pulp necrosis than teeth in the Orthodontics
group (P < .001) or the corresponding teeth in the Trauma group (P = .004). No significant differences were observed between teeth in the Orthodontics/Trauma
group with previous hard tissue injuries and teeth in the Orthodontics group or the
corresponding teeth in the Trauma group. In addition, no statistically significant
differences were determined between central and lateral incisors.
Conclusions
The results indicated that maxillary incisors with a history of severe periodontal
injury have a higher susceptibility to pulp necrosis during orthodontic extrusion
than nontraumatized teeth.
Key Words
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Article info
Publication history
Published online: December 14, 2009
Identification
Copyright
© 2010 American Association of Endodontists. Published by Elsevier Inc. All rights reserved.